10 Things I Wish People Knew About Tourette Syndrome

By Lucy Clapham

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I have lived with Tourette syndrome (TS) for most of my life, but I was only diagnosed when I was 17. Technically I was told when I was 15, but I laughed it off due to my own ignorance about the condition. So for the benefit of those who are facing a TS diagnosis themselves or are just interested, let me tell you some lesser-known things about TS.

1. There is more to Tourette than swearing.

I cannot stress this enough. Although coprolalia, the fancy word for involuntary swearing, can be a symptom, it is surprisingly rarer than the majority of television shows would have you believe. Only an estimated 10 to 15 percent of people with Tourette syndrome swear involuntarily, me being one of them.

2. We can’t “just stop it.”

If only it was that easy! Yes, I know it’s an annoying noise and this really isn’t the best place to be making said noise, but I literally cannot stop! Many of us get what is known as a premonitory sensation or urge that can feel like an itch inside the body or a small jolt of electricity. Some of us can, and do, learn to suppress our tics, but it can be hard for us to do this. To help you understand, try not to blink. That horrid feeling that builds up in the back of your eyes — that’s what we get all over our bodies! Just as you will need to blink, we will eventually need to tic, and it can sometimes make it worse. So don’t ask us to suppress our tics!

3. It can be painful.

Although many tics are harmless, some can cause either minor or severe pain. This can be due to repetitive movements, punching or kicking things, biting and scratching ourselves and in my case, even running myself into walls at breakneck speed. If I injure one of my joints, my TS will focus on the injured limb and I won’t stop twisting it.

4. Some people have tic “storms” or fits.

Some of my friends in the Tourette community and I suffer from what we call “tic fits” or “storms” where we completely lose control of our bodies to tics. This can be scary and painful for us, and I believe it’s a poorly researched part of TS by professionals.

5. Laughing is fine… sometimes.

While we do have a sense of humor, you really need to know when it’s OK to laugh and when it isn’t. Generally speaking, my rule is “If I’m laughing, you may laugh, too.” It is extremely difficult not to laugh when I’ve shouted “Donkey Kong dropkicked a tortoise!” at random. If I am obviously not happy, don’t laugh! Always ask the individual with TS whether laughing is OK and when.

6. It often comes with “added extras.”

Many people don’t just have TS. Quite a lot of us also have obsessive compulsive disorder, attention deficit hyperactivity disorder, autistic spectrum disorder and various other issues, often to do with learning. I couldn’t write until I was 12 years old; others may struggle with reading or math.

7. Some of us don’t grow out of it.

Although a lot of doctors say that most people grow out of TS at around 18 years old, this isn’t always the case and about 5 to 10 percent of people with TS continue to have symptoms as adults.

8. Some activities can calm tics.

Some of us find that our tics reduce or disappear completely when we are engaged in certain activities, such as playing a musical instrument, jogging or other forms of exercise, playing computer games or spending time with an animal. Everyone is different in this respect, and I expect some of us don’t have a special activity, but some do. For me, it’s playing the guitar.

9. Pointing out tics can make them worse.

I can promise you that we are aware we are making noise or doing a strange movement. Pointing this out not only causes more embarrassment, but, for me, can also make me feel more like I need to do the tic. My dad once thought it would be helpful to point out that I was squeaking, but instead of stopping it, I could no longer control it!

10. There is no cure.

There is no cure at this moment. The only options for treatment include medications or, if the TS is severe, deep brain stimulation. Neither is a cure for TS but may reduce the severity of the symptoms. I don’t take medication for my TS because the side effects outweigh the benefits for me.

Source: TheMighty.com

New drug target for Rett syndrome


By Hannah Robbins

Harvard Stem Cell Institute (HSCI) researchers have identified a faulty signaling pathway that, when corrected in mice, ameliorates the symptoms of Rett syndrome, a devastating neurological condition. The findings could lead to the discovery of compounds or drugs that may benefit children affected by the disease, says neurobiologist Jeffrey Macklis, a member of the HSCI Executive Committee.

The research was published recently in Nature Communications. Noriyuki Kishi and Jessica MacDonald, both recent postdoctoral fellows in the Macklis laboratory, are co-first authors. Macklis, who directed the work, is the Max and Anne Wien Professor of Life Sciences in the Department of Stem Cell and Regenerative Biology, and Center for Brain Science at Harvard University.

Rett syndrome is a relatively common neurodevelopmental disorder, the second most common cause of intellectual disability in girls after Down syndrome; it is associated with a dysfunctional gene on the X chromosome. Boys with Rett syndrome are rare, because male fetuses who carry the mutations on their one X chromosome usually have prenatally lethal forms of the disease.

Girls with Rett syndrome appear to develop relatively normally for the first six to 18 months of life, but then regress; they tend to lose their ability to speak and the purposeful use of their hands, withdraw from social situations, and wring their hands.

Austrian physician Andreas Rett first described the disorder in 1966, but it wasn’t until 1999 that Huda Zoghbi and her lab at Baylor College of Medicine identified mutations in the gene MECP2 as the root cause of Rett syndrome. MECP2, however, turns a very large number of genes on and off throughout the entire body, so it has been a long-standing puzzle why children with Rett syndrome have this very specific and reproducible developmental cognitive brain disorder.

“My view was that MECP2 mutation in Rett syndrome disrupts so many genes and their protein products that we weren’t going to find a single gene that we could fix to help girls with Rett,” said Macklis, former program head of HSCI’s Nervous System Diseases Program, and an Allen Distinguished Investigator of the Paul G. Allen Family Foundation. “But if we found a disrupted, improperly regulated signaling pathway that was ‘drug-able,’ that affected enough of the girls’ pathology, we might be able to make them dramatically functionally better with already available therapeutics — and that might make a real difference in their lives and their families’ lives.”

Instead of concentrating on the MECP2 gene, Macklis’ group focused on neurons he knew were “abnormal and implicated in Rett syndrome and autism spectrum disorders,” and in 2004, his lab was the first to describe abnormal development in this type of neuron. These neurons, called inter-hemisphericcallosal projection neurons (CPNs), have shorter, less-developed dendrites, or “receiving antennas,” in mice with the Rett gene mutations and in individuals with Rett syndrome.

Building on their 2004 findings, the researchers were able to fluorescently label CPNs in mice with or without the Rett mutation, purify them from other types of neurons, and look at the levels at which many thousands of genes were active, and thus how much of the proteins coded for by those those genes was made.

They found that one gene for IRAK1, which Macklis’ group identified as regulated by MECP2 and which is a well-known part of the NF-kB signaling pathway, was making about three times more protein than normal. They modified IRAK1 levels both in mice with Rett mutations and in mouse neurons in culture dishes. When they reduced the activity of its gene Irak1 by roughly half, and consequently the amount of IRAK1 protein made, the neurons and their dendrites developed substantially better, indistinguishable from normal by several assays. Further, mice with reduced levels of IRAK1 had significantly fewer symptoms, better function, and much longer lifespan. They had much-improved health, well beyond only these neurons.

Now, Macklis said, the researchers have started looking into potential compounds and drugs that are already available and that might partially correct this pathway, and what dosages and timing might ultimately ameliorate the effects of Rett syndrome.

Source: News.Harvard.edu

Meet the Woman Who Cared for Hundreds of Abandoned Gay Men Dying of AIDS


We Cannot Continue to Overlook ‘High-Functioning’ Depression

digital painting of sketched beautiful girl in hat, acrylic on canvas texture

By Amanda Leventhal

I first saw a psychiatrist for my anxiety and depression as a junior in high school. During her evaluation, she asked about my classes and grades. I told her that I had a 4.0 GPA and had filled my schedule with Pre-AP and AP classes. A puzzled look crossed her face. She asked about my involvement in extracurricular activities. As I rattled off the long list of groups and organizations I was a part of, her frown creased further.

Finally, she set down her pen and looked at me, saying something along the lines of, “You seem to be pretty high-functioning, but your anxiety and depression seem pretty severe. Actually, it’s teens like you who scare me a lot.”

Now I was confused. What was scary about my condition? From the outside, I was functioning like a perfectly “normal” teenager. In fact, I was somewhat of an overachiever. I was working through my mental illnesses and succeeding, so what was the problem?

I left that appointment with a prescription for Lexapro and a question that I would continue to think about for years. The answer didn’t hit me all at once; rather, it came to me every time I heard a suicide story on the news saying, “by all accounts, they were living the perfect life.” It came to me as I crumbled under pressure over and over again, doing the bare minimum I could to still meet my definition of success. It came to me as I began to share my story and my illness with others, and I was met with reactions of “I had no idea” and “I never would have known.”

It’s easy to put depression into a box of symptoms, and though we as a society are constantly told mental illness comes in all shapes and sizes, we are stuck with a mental health stock image in our heads that many people don’t match. When we see depression and anxiety in adolescents, we see teens struggling to get by in their day-to-day lives. We see grades dropping. We see involvement replaced by isolation. People slip through the cracks.

We don’t see the student with the 4.0 GPA. We don’t see the student who’s active in choir and theater or a member of the National Honor Society. We don’t see the student who takes on leadership roles in a religious youth group. No matter how many times we are reminded that mental illness doesn’t discriminate, we revert back to a narrow idea of how it should manifest, and that is dangerous.

Recognizing that danger is what helped me find the answer to my question. Watching person after person, myself included, slip under the radar of the “depression detector” made me realize where that fear comes from. My psychiatrist knew the list of symptoms, and she knew I didn’t necessarily fit them. She understood it was the reason that, though my struggles with mental illness began at age 12, I didn’t come to see her until I was 16. Four years is a long time to deal with mental illness alone, and secondary school is a dangerous time to deal with it.

If we keep allowing our perception of what mental illness looks like to dictate how we go about recognizing and treating it, we will continue to overlook those who don’t fit the mold. We cannot keep forgetting that there are people out there who, though they may not be able to check off every symptom on the list, are heavily and negatively affected by their mental illness. If we forget, we allow their struggle to continue unnoticed, and that is pretty scary.

Source: TheMighty.com

Why And How To Cultivate Optimal Thyroid Health

Daniel Haun
by Daniel Haun

The thyroid hormones help all your organs work well. Learn why it is important to cultivate optimal thyroid health and how to heal your thyroid naturally.

The thyroid gland is shaped like a butterfly and is found at the front of the neck, below the Adam’s apple.

Although small in size, this gland has a big influence on the body’s ability to transform food into the energy needed to sustain life.

Why Is Your Thyroid Important?

Thyroid hormones control the growth, temperature, and function of every cell in the body.

It is a vital component of the endocrine system, an elaborate network of hormone-producing glands acting upon the cardiovascular, digestive, neurological, and reproductive systems.

The importance of this gland, therefore, cannot be understated!

When functioning properly, the thyroid gland secretes just the right amount of thyroid hormone to regulate almost all the metabolic processes in your body.

Too much or too little of these vital body chemicals, and it can drastically influence energy levels, body weight, and your mental health.

Cultivating your thyroid health, combined with proper nutrition and diet, can boost energy, improve appetite, reduce insomnia, relieve depression symptoms, improve circulation, relieve muscle aches, and assist you in recovering from endocrine disorders.

How To Heal Your Thyroid Naturally

An easy way to improve thyroid health and the health of your endocrine system, is to eat nutritious meals and maintain a well-balanced diet.

There are two primary ways the thyroid can malfunction: It can be overactive, or hyperthyroid, producing symptoms such as a rapid heartbeat, excessive hunger, weight loss, diarrhea, and heat sensations.

Or, conversely, it can underproduce, known as hypothyroid, creating symptoms like a slow heartbeat, reduced appetite, weight gain, constipation, and cold sensations.

If you experience any symptoms, it is important to see your medical doctor in addition to visiting your practitioner of acupuncture and Oriental medicine.

Hyperthyroidism and hypothyroidism can be diagnosed by testing the levels of thyroid hormones in your blood.

Hormones secreted by the thyroid are measured, as well as thyroid-stimulating hormone (TSH), a chemical released by the pituitary gland that triggers hormone production in the thyroid.

Oriental medicine treatments take all symptoms into account and are aimed at balancing the production and release of thyroid hormones through a variety of approaches, including acupuncture and herbal remedies, lifestyle changes, and special exercises.

In the treatment of thyroid problems, acupuncture can be used to restore hormonal balance, regulate energy levels, smooth emotions, and help manage sleep.

There are several acupuncture points on the ear and the body that can be used to regulate the production of thyroid hormones.

Source: Curejoy.com


Multiple Sclerosis is close to being cured after a pioneering treatment was found to stop and even reverse the disease, scientists have said.

Results of a trial were hailed as remarkable, with the progression of the debilitating disease halted in almost all patients who had the treatment. A quarter of the MS sufferers had their condition effectively suppressed.

In the trial at the University of Ottawa in Canada, patients had aggressive chemotherapy combined with a transplant of their own cells. The chemotherapy destroyed the immune system instead of suppressing it as in standard treatment. It was then “reset” using blood stem cells.

The new technique is not without risk. Of the 24 patients treated, one died as a consequence, and the study was too small to assess the true dangers. Even so, the treatment was described as a breakthrough therapy which was “close to being curative”.

MS is a chronic inflammatory disease of the central nervous system. It is caused when the immune system attacks the body and can result in vision problems, memory loss, dizziness, fatigue and spasms. In severe cases it can lead to paralysis.

The condition has very few treatment options and often patients live for many years with the debilitating symptoms.

One of those treated in the Ottawa trial was Jennifer Molson, who was diagnosed with MS in 1996 when she was 21. She had an aggressive form of the condition and limited mobility when she began the trial, and received her transplant in 2002.

“Before my transplant I was unable to walk or work and was living in assisted care at the Ottawa hospital rehabilitation centre,” she said. “Now I am able to walk independently, live in my own home and work full time. I was also able to get married, walk down the aisle with my dad and dance with my husband. I’ve even gone downhill skiing. Thanks to this research I have been given a second chance at life.”

Researchers enrolled 24 people aged 18 to 50 with aggressive MS who had not responded to immunosuppressive therapy. They were followed for an average of six-and-a-half years to test the long-term effects of chemotherapy followed by the treatment, called autologous haematopoietic stem cell transplantation (aHSCT).

After treatment 70 per cent of patients experienced a complete stop in disease progression and 40 per cent saw lasting reversal of symptoms. Several were able to return to work or school and have children. None needed medication.

“[This shows with] unprecedented clarity a near-complete suppression of MS disease for several years following aHSCT,” Paolo Muraro, of Imperial College London, said. “None of the drugs that are currently approved or in late-phase clinical trials has been reported to achieve a similarly profound control of the disease.”

Stephen Minger, a stem cell biologist and independent consultant, said: “The clinical results are truly impressive, in some cases close to being curative, though we need longer-term follow-up to know for certain whether the patients continue to do well or if there is a chance of relapse. For a lifelong progressive disease like MS with few treatment options this is really exciting data.”

During treatment one of the patients died from hepatic necrosis and sepsis caused by the chemotherapy, which is one reason clinicians are approaching the results with caution.

Harold Atkins, a stem cell transplant physician at the Ottawa hospital and author of the study, said: “Our trial is the first to show the complete, long-term suppression of all inflammatory activity in people with MS. However, it is important to note that this therapy can have serious side effects and risks, and would only be appropriate for a small proportion of people with very active MS.”

In Britain MS sufferers can receive aHSCT treatment only if they are chosen for a clinical trial. Emma Gray, a clinical lead at the MS Society, said that more research was needed. “This treatment does offer hope, but it’s also an aggressive procedure that comes with substantial risks and requires specialist after-care. If anyone is considering aHSCT we would recommend they speak to their neurologist,” she said.

Photo of F.S.U. Football Star Sitting With Boy Eating Alone at School Charms Internet


Travis Rudolph, a star football player at Florida State University in Tallahassee, is no stranger to the spotlight. He is famous for his award-winning athleticism, catching high and running far as a wide receiver.

But it was Mr. Rudolph’s action off the football field that earned him national attention this week when, visiting a local school on a good-will visit with his teammates on Tuesday, he made a simple gesture.

He sat down at a lunch table.

In a moment captured on camera and shared widely on social media, Mr. Rudolph saw a boy sitting alone in the cafeteria at Montford Middle School.

“So I asked him could I sit down and have lunch with him,” Mr. Rudolph said in an interview with “Fox and Friends” on Thursday. “And he said, ‘Sure why not?’ ”

The boy introduced himself. His name was Bo Paske. He was a sixth grader.

“And the conversation went from there,” Mr. Rudolph said.

Although Mr. Rudolph, 21, did not know it at the time, Bo has autism and often eats lunch by himself, according to the boy’s mother, Leah Paske, who wrote about the moment and published the photograph on Facebook on Tuesday. Since then, the image of the football star eating pizza while seated opposite Bo has became an example of how a small act of kindness can go big.

Mr. Rudolph, Bo and Ms. Paske have since been inundated with requests for interviews with national media organizations, appearing on news programs and in newspapers. Ms. Paske’s post has been shared more than 13,000 times, and the photograph has been circulating on Twitter, including on the account of Autism Speaks, an advocacy and support organization, from where it was retweeted nearly 600 times.

“I did not even recognize that it will be this big,” Mr. Rudolph told the Fox show as Bo sat next to him and his mother sat on the other side of her son. “Everybody is the same, and one man can make a difference.”

Efforts to reach Mr. Rudolph through a family member were unsuccessful on Thursday. Ms. Paske did not immediately return emails and phone calls seeking comment.

The story also shed light on some of the challenges that face families coping with relatives who have autism spectrum disorder, which the Centers for Disease Control and Prevention defines as a group of developmental disabilities that can cause social, communication and behavioral challenges in about one in 68 children.

Ms. Paske said in her Facebook post that she was sometimes “grateful” that her son had the condition.

“That may sound like a terrible thing to say, but in some ways I think, I hope, it shields him,” she wrote. “He doesn’t seem to notice when people stare at him when he flaps his hands. He doesn’t seem to notice that he doesn’t get invited to birthday parties anymore. And he doesn’t seem to mind if he eats lunch alone.”

She said that she asks him questions about his school experience every day.

“ ‘Was there a time today you felt sad?’ ‘Who did you eat lunch with today?’ Sometimes the answer is a classmate, but most days it’s nobody. Those are the days I feel sad for him, but he doesn’t seem to mind. He is a super sweet child, who always has a smile and hug for everyone he meets.”

She said that a friend of hers was at the school on the day that Mr. Rudolph and his teammates visited.

“I am not sure what exactly made this incredibly kind man share a lunch table with my son, but I’m happy to say that it will not soon be forgotten,” Ms. Paske said on Facebook. “This is one day I didn’t have to worry if my sweet boy ate lunch alone, because he sat across from someone who is a hero in many eyes.”

During the interview on Thursday, Ms. Paske recounted how anxious she had been as Bo attended middle school. As she spoke, Bo occasionally looked up at her and reached over to touch her arm whenever he saw that she was distressed.

Bo said that it was “amazing” that Mr. Rudolph had chosen to sit down with him, adding that the player had even signed his lunchbox.

Asked how the encounter had made him feel, Bo said, “It was kind of like me sitting on a rainbow.”

Source: Nytimes.com

What You Can’t Always See About Living With Parkinson’s Disease

By Jean Mellano

Businesswoman with to go coffee looking in purse at cafe.

In November of 2015, I was diagnosed with Parkinson’s disease (PD). I had told my primary care doctor  I was having problems writing. She referred me to a neurologist who, after some movement tests, told me I had PD.

Now other strange things that had been occurring in my body seemed to add further fuel to that diagnosis. I was having problems cutting my salad with the fork in my left hand. My flip flop sandals and clogs would not stay on my left foot when climbing steps. Flossing my teeth and washing my hair using my left hand are now challenging to say the least. From someone who used to type 60 words a minute with no errors, I now hunt and peck on the keyboard. My balance, flexibility and fluidity of movement, which used to be excellent from all my years of dance is failing. Swallowing sometimes is difficult. When I roll over in bed at night, sometimes it is so arduous, I wish I had a forklift to help me. Putting items into a bag or finding credit cards and money in my wallet confound me at times.

Although they are constant reminders I have a progressive, non-curable disease, these simple tasks I can no longer do with ease, things I have always taken for granted, are just daily aggravations and annoyances. Luckily I was a problem solver for most of my career so this skill comes in handy now. Most people look at me and say I look fine and nothing has changed.

However, there are two symptoms no one sees that really affect the quality of my life the most.

The first is the slowness of movement: it takes me forever to get dressed or to make
something to eat. Putting together a salad seems to take an eternity. Needless to say, I don’t eat much.

The second symptom and the most life-impacting is the extreme fatigue. This is a fatigue that cannot be diminished by rest or sleep. I wake up after a sound night’s sleep totally exhausted. Since I am so tired, being motivated to exercise no longer exists for me.
To bike 10 miles now (this used to be a warm up for me) takes me almost an hour, and I am wiped out after it. Most days, I will just lay in bed for hours in the afternoon since I am so exhausted after I have forced myself to exercise in the morning.

If it were not for the discipline and muscle memory I developed from years of dance and exercise, I might be in a far worse off situation. Since I have such bad side effects from the prescribed medications, I must rely on continued movement and exercise to help me battle this disease.

So, one day, if you see someone moving slow and you are in a hurry and starting to get annoyed since this person is holding up the line, that person might be someone with PD trying to find a credit card in their wallet. Try to have a little more patience and compassion.

Source: Themighty.com

Children with autism could soon be prescribed medical marijuana

– The Minnesota Department of Health is considering adding autism to the growing list of conditions in which the patients can be prescribed medical cannabis.

The consideration carries with it a lot of emotion for families looking for something to help their loved ones with a condition where so little is known.

In a life full of uncertainty, Kammy Krammer is sure of one thing: medical cannabis helps her autistic son, Elliot, in profound way. But 15-year-old Elliott’s autism is not why he’s allowed under state law to take it.

“Elliott suffered from debilitating anxiety and that affected every aspect of his life,” Kammy told Fox 9. “He had hundreds of tics every single day and was suffering with peer relationships because of that. He was embarrassed and he couldn’t control them.”

Kammy said Elliot started using medical cannabis July 1, 2015 – the first day it was legal in Minnesota – to help manage his Tourette syndrome. He had an immediate 90 percent reduction in tics, but his mom also started to notice it was also helpful for many of the side effects related to autism.

Tourette’s, not autism, is one of a handful of conditions in Minnesota where medical cannabis can be administered.

Now, the DOH is considering adding autism to the list, but in that consideration, they really only have anecdotal evidence from parents like Kammy and kids like Elliott that it actually works, creating a degree of “uncertainty” around an already mysterious condition with no cure.

Autism is the result of alterations in how the brain processes information which then alters how the mind sees the world. For someone with autism, that often means communication problems, social challenges and repetitive behaviors.

As for how it’s leaning on the decision, the DOH issued a statement saying the process needs to take its course before we comment on specific proposals. The state’s medical cannabis review panel will report on the public health benefits and risks of any proposed medical conditions by Nov. 1.

Jonah Weinberg is the director of the Autism Society of Minnesota. He says parents are challenged daily to find something that works for their child. But, giving cannabis, even the medical kind, needs to be considered carefully.

“What we hope is that they are approached reasonably, that they’re approached methodically and that it’s not just anecdotal evidence,” Weinberg said. “That one family says, ‘Oh, this worked for my child,” because a case study of one does not mean anything in the long term.”

On the “anecdotal evidence” side, Elliott shines. A year on medical cannabis for his Tourette’s and the subsequent effect on his autism have allowed him to be mainstreamed at school and he is now running on the Eagan High School cross country team.

While he is a young man of few words, Elliot told Fox 9 being a part of the simplest of teenage things makes him feel whole.

A year ago, Kammy could not have imagined her son attending a high school football game or running with his teammates on the track. Now, she wants that for other kids like Elliot.

“I believe it is worth fighting for because I know Elliott is not alone. I know that there are other kiddos with autism and their families and believe it should be their option to explore,” Kammy said.

In addition to autism, the department of health is considering things like arthritis, depression, diabetes, insomnia and PTSD as conditions that should be allowed to be treated with medical cannabis. A decision on whether to add autism or conditions will likely come next month.

Source: Fox9.com

Former Horse Jumper With Parkinson’s Disease Gets Her Wish to Ride One More Time

An 87-year-old former horse jumper with Parkinson’s disease got her wish to ride again.

Nelly Jacobs, of the Netherlands, was diagnosed with the debilitating disease more than three decades ago, forcing her to give up riding. Now she spends most her days in a wheelchair in hernursing home.

But, thanks to Hidden Desires, a project of two charities called The Care Group and the Green Cross, Jacobs was granted her wish to ride a horse again at a nearby riding school.

“Horses have always been important to her; ever since she was a child, until her Parkinson’s got the best of her,” Jacobs’ son Jan said in a now viral Facebook video.

He added, “Her uncle Fons put her on a horse when she was 9. Later on, she rode several horses and she even made a decent jumper.”

Jan accompanied his mother to the riding school where she was able to meet several horses in a stable and even smell the hay, an aroma she called “delicious,” before riding.

“This is a very emotional day for her,” her son said. “So she won’t be able to speak, but I can tell she loves every second of seeing the horses, smelling the stables,” her son said. “These things matter to her because she must have missed it at the nursing home.”

After her meet-and-greet, Jacobs was brought to an enclosed arena. She was then gently lifted onto the backs of two horses, who rode side-by-side.

Jacobs called the experience “wonderful” as a broad smile appeared on her face.

“Lots of memories are coming back to her,” her son said in the video. “Her whole life revolved around horses, so through this experience, she’s able to feel the joy again and reminisce about things from her past.”

Source: Yahoo.com