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A mysterious polio-like illness that paralyzes people may be surging this year

Before dinner on July 29, 3-year-old Carter Roberts of Chesterfield, Va., seemed perfectly healthy. That evening, he vomited. When he woke up the next morning with a slight fever of 99 degrees, his mother, Robin Roberts, figured that he was coming down with a cold. The next morning, she found him collapsed on his bedroom floor.

“Mommy,” she recalls him saying. “Help me, help me.”

Carter could barely stand when she picked him up, and his neck was arched backward. “What was most alarming,” she said, “is he had no control over his right arm whatsoever.”

In the hospital, Carter lost control of his right arm, then over his legs and other muscles within a few days. He now can only wiggle a toe and move the left side of his face. He has been diagnosed with a mysterious, polio-like illness called acute flaccid myelitis, a condition that seems to be surging this year.

Through July, 32 new cases of AFM have been confirmed across the United States this year by the Centers for Disease Control and Prevention, a sharp rise compared with last year, when just seven cases had been confirmed by that month. The numbers have risen steadily since April. In past years, most cases have occurred between August and December, with a peak in October.

Among the many unanswered questions about the condition are what causes it, how best to treat it and how long the paralysis lasts. Although most cases occur in children, AFM occasionally affects adults.

The CDC official who leads the surveillance efforts said that confirmed numbers for August will not be available until the end of this month, but the number of reports she is receiving from doctors around the country continues to rise.

“CDC is looking at these trends very carefully,” Manisha Patel said. “We have sent out several health alerts to states to let them know we are seeing an increase in reporting and to encourage them to communicate with doctors to report these cases in a timely fashion.”

The CDC began tracking AFM in 2014, when 121 cases were confirmed. That year, the CDC counted only children affected by the disease. Their average age was 7. Most had a fever or a respiratory illness a few days before developing paralysis. Many had to be placed on respirators. Although 85 percent of the children recovered partially, only three of them recovered fully.

Mom celebrates as son with AFM sits up unassisted

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Five-year-old Braden Scott sits up unassisted at a rehab facility two months after he was struck with acute flaccid myelitis. It’s a major improvement for someone with this polio-like illness. (Facebook/Team Braden Luke)

Health officials and physicians around the country said they are concerned that the rising number of cases through July could foretell a repeat of 2014.

“You hate to be an alarmist, but there’s reason to have some concern,” said Avindra Nath, chief of the section of infections of the nervous system at the National Institute of Neurological Disorders and Stroke. “What we don’t know is where are these cases. Are they clustered? Do they all look alike?  Getting more information on these cases would be helpful.”

Beyond saying that confirmed cases have been reported in 17 states through July, Patel declined to reveal which states have been involved. But conversations and emails with physicians around the country indicate that at least four cases have occurred in California and at least three in Massachusetts, and that others have been seen in Florida, Texas, Pennsylvania and New York, as well as Virginia.

“We are definitely hearing of cases from our colleagues across the country,” said Teri Schreiner, a neurologist at the University of Colorado and Children’s Hospital Colorado. “It’s a trend that’s worrisome … what I’m hearing from others seems to be coming at a tempo similar to what happened in 2014.”

Jean-Baptiste Le Pichon of Children’s Mercy Hospital in Kansas City, Mo., said that doctors on a U.S. email list for pediatric neurologists reported five new cases of AFM in just the past few days. “There is definitely an explosion of cases,” Le Pichon said.

No treatment other than physical therapy has yet been shown to markedly improve outcomes, although some doctors have reported that treatment with intravenous immunoglobulin (IVIG), used to treat some other viral infectious diseases afflicting the nervous system, might help. It’s also been suggested that Prozac could prove beneficial. The degree of paralysis in the first month has generally improved only slightly over the course of a year.

“After about a year or so, what you’ve got is what you’ve got,” said Max Wiznitzer, a pediatric neurologist at Case Western Reverse University in Cleveland.

Most perplexing is what causes the disease. The 2014 outbreak of AFM occurred at the same time as a far larger outbreak of enterovirus D68 across the United States. The vast majority of patients infected with the virus developed only a respiratory illness. Some physicians were convinced that EV-D68 was the cause, not only because both outbreaks occurred at the same time but also because of a study that identified a particular strain of EV-D68 in the airways of children with AFM. But officials at the CDC and some doctors, including Wiznitzer, insist that the cause remains unproved.

So far this year, no similarly widespread outbreak of EV-D68 respiratory infections has been reported, although cases of it and other enteroviruses have been seen in some areas.

On Friday, Le Pichon, in Kansas City, said, “I just got confirmation that we have an epidemic of enterovirus breaking out here and at least a few cases [of enterovirus] have typed positive for EV-D68.”

Carol Glaser, a pediatric infectious-disease specialist at Kaiser Permanente in Oakland, Calif., said: “If the theory is right that it’s caused by EV-D68, which some of us believe more than others, this would be the time of year we start seeing it. It’s an enterovirus, and they tend to peak in late summer and early fall.”

Because the CDC’s surveillance of AFM is less than two years old, it remains unclear if some of this year’s apparent rise in confirmed cases is due simply to more doctors sending in case reports, Patel said. And Wiznitzer emphasized that with just 32 confirmed cases across the United States so far, AFM remains extremely rare

Glaser, however, said she is convinced that the condition is new. She worked at the California Department of Public Health when, in 2012, some of the first reports of AFM anywhere in the country came in from physicians there.

“I was there for 15 years and talked to neurologists every single day,” she said. “It wasn’t until the fall of 2012 when the first physician called in and wanted a test for polio on one of his patients. One of my colleagues in the office chuckled and said to me, ‘We don’t have polio in the United States.’ But in the next two weeks, I got two very similar phone calls. We had never had a report like that. I do think there was something new going on. Fortunately, it’s still rare.”

If EV-D68 does somehow cause AFM, studying the virus may lead to prevention or treatment. One glimmer of good news came from Kenneth Tyler, chair of neurology at the University of Colorado School of Medicine. He and his colleagues have been conducting experiments to infect mice with EV-D68 and have them develop loss of muscle control like that seen in AFM. He says he has submitted a paper to a peer-reviewed medical journal describing his success in doing so.

The goal, he said, is to use mice to study how EV-D68 causes harm to the spinal cord and muscles, and to test potential therapies, such as IVIG.

“It looks like we’re seeing protection with IVIG in the mouse model,” he said.

Robin Roberts said she wishes more was known about the condition that has harmed her son. “In this day and age to not have information about something like this is very frustrating. There needs to be more done about it, and for more parents and doctors to know about it,” she said.

Angie Andersen said that her daughter McKenzie was sent home from the hospital twice in the first two weeks of her illness because doctors did not understand the severity of what was happening. Finally her pediatrician told Andersen to bring McKenzie to another hospital.

McKenzie was 6 years old when she developed sniffles in December 2014. “Within 12 days,” Andersen said, “she was paralyzed from the neck down, on a ventilator to breathe for her. She was left with her left hand and her feet and toes that move.”

“You know you could understand the bad that comes from a car wreck, or if she got cancer,” Andersen said. “How do you ever wrap your brain around the fact that she got a cold, and now she’s a quadriplegic on a ventilator? It’s a nightmare you never wake up from.”

Still, she said, McKenzie can now speak and even sing softly despite having a tracheotomy. “She has a lot of spunk, and that is helping her through,” Andersen said.

SOURCE: washingtonpost

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Oh See What’s Inside the adult ADHD brain?

Brain scans differentiate adults who have recovered from childhood ADHD and those whose difficulties linger.At left, the brains of adults who had ADHD as children but no longer have it show synchronous activity between the posterior cingulate cortex (the larger red region) and the medial prefrontal cortex (smaller red region). At right, the brains of adults who continue to experience ADHD do not show this synchronous activity.
Illustration: Jose-Luis Olivares/MIT (based on images courtesy of the researchers)

About 11 percent of school-age children in the United States have been diagnosed with attention deficit hyperactivity disorder (ADHD). While many of these children eventually “outgrow” the disorder, some carry their difficulties into adulthood: About 10 million American adults are currently diagnosed with ADHD.

In the first study to compare patterns of brain activity in adults who recovered from childhood ADHD and those who did not, MIT neuroscientists have discovered key differences in a brain communication network that is active when the brain is at wakeful rest and not focused on a particular task. The findings offer evidence of a biological basis for adult ADHD and should help to validate the criteria used to diagnose the disorder, according to the researchers.

Diagnoses of adult ADHD have risen dramatically in the past several years, with symptoms similar to those of childhood ADHD: a general inability to focus, reflected in difficulty completing tasks, listening to instructions, or remembering details.

“The psychiatric guidelines for whether a person’s ADHD is persistent or remitted are based on lots of clinical studies and impressions. This new study suggests that there is a real biological boundary between those two sets of patients,” says MIT’s John Gabrieli, the Grover M. Hermann Professor of Health Sciences and Technology, professor of brain and cognitive sciences, and an author of the study, which appears in theJune 10 issue of the journal Brain.

Shifting brain patterns

This study focused on 35 adults who were diagnosed with ADHD as children; 13 of them still have the disorder, while the rest have recovered. “This sample really gave us a unique opportunity to ask questions about whether or not the brain basis of ADHD is similar in the remitted-ADHD and persistent-ADHD cohorts,” says Aaron Mattfeld, a postdoc at MIT’s McGovern Institute for Brain Research and the paper’s lead author.

The researchers used a technique called resting-state functional magnetic resonance imaging (fMRI) to study what the brain is doing when a person is not engaged in any particular activity. These patterns reveal which parts of the brain communicate with each other during this type of wakeful rest.

“It’s a different way of using functional brain imaging to investigate brain networks,” says Susan Whitfield-Gabrieli, a research scientist at the McGovern Institute and the senior author of the paper. “Here we have subjects just lying in the scanner. This method reveals the intrinsic functional architecture of the human brain without invoking any specific task.”

In people without ADHD, when the mind is unfocused, there is a distinctive synchrony of activity in brain regions known as the default mode network. Previous studies have shown that in children and adults with ADHD, two major hubs of this network — the posterior cingulate cortex and the medial prefrontal cortex — no longer synchronize.

In the new study, the MIT team showed for the first time that in adults who had been diagnosed with ADHD as children but no longer have it, this normal synchrony pattern is restored. “Their brains now look like those of people who never had ADHD,” Mattfeld says.

“This finding is quite intriguing,” says Francisco Xavier Castellanos, a professor of child and adolescent psychiatry at New York University who was not involved in the research. “If it can be confirmed, this pattern could become a target for potential modification to help patients learn to compensate for the disorder without changing their genetic makeup.”

Lingering problems

However, in another measure of brain synchrony, the researchers found much more similarity between both groups of ADHD patients.

In people without ADHD, when the default mode network is active, another network, called the task positive network, is suppressed. When the brain is performing tasks that require focus, the task positive network takes over and suppresses the default mode network. If this reciprocal relationship degrades, the ability to focus declines.

Both groups of adult ADHD patients, including those who had recovered, showed patterns of simultaneous activation of both networks. This is thought to be a sign of impairment in executive function — the management of cognitive tasks — that is separate from ADHD, but occurs in about half of ADHD patients. All of the ADHD patients in this study performed poorly on tests of executive function. “Once you have executive function problems, they seem to hang in there,” says Gabrieli, who is a member of the McGovern Institute.

The researchers now plan to investigate how ADHD medications influence the brain’s default mode network, in hopes that this might allow them to predict which drugs will work best for individual patients. Currently, about 60 percent of patients respond well to the first drug they receive.

“It’s unknown what’s different about the other 40 percent or so who don’t respond very much,” Gabrieli says. “We’re pretty excited about the possibility that some brain measurement would tell us which child or adult is most likely to benefit from a treatment.”

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Monty Python’s Terry Jones diagnosed with dementia

Monty Python star Terry Jones has been diagnosed with a severe variant of dementia.

The 74-year-old has primary progressive aphasia, which affects his ability to communicate.

As a result, Jones “is no longer able to give interviews”, his spokesman said.

The news was confirmed as Bafta Cymru announced the Welsh-born comedian is to be honoured with an outstanding contribution award.

The National Aphasia Association describes primary progressive aphasia as a neurological syndrome in which language capabilities become slowly and progressively impaired.

“It commonly begins as a subtle disorder of language, progressing to a nearly total inability to speak, in its most severe stage,” their website states.

Image caption     Left-right: Terry Jones, Graham Chapman, John Cleese, Eric Idle, Terry Gilliam and Michael Palin

Jones, who is from Colwyn Bay in north Wales, was a member of the legendary comedy troupe with Terry Gilliam, John Cleese, Eric Idle, Michael Palin and the late Graham Chapman.

He directed Monty Python’s Life of Brian and The Meaning of Life and co-directed Monty Python and the Holy Grail with Gilliam.

The surviving members reunited for 10 reunion performances at the O2 Arena in London in 2014.

Kathryn Smith, director of operations at Alzheimer’s Society, said: “We are deeply sorry to hear about Terry Jones’s diagnosis of dementia and are thinking of Terry and his family during this time.”

Image caption   Jones and the surviving members of the original Monty Python cast attended the Tribeca Film Festival last year

His award was announced at the Bafta Cymru nominations party, ahead of the British Academy Cymru Awards on 2 October.

“Terry is proud and honoured to be recognised in this way and is looking forward to the celebrations,” his spokesman said.

Hannah Raybould, director of Bafta Cymru, said: “We are very much looking forward to celebrating the work of Terry Jones during the ceremony with a look back at his work from 1969 to the present day.”

source:  BBC.com

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British Man Is First To Recover From HIV, Gives Hope To Millions Suffering From The Disease

The 44-year-old British man is actually the first person in the history to be cured of HIV! Scientists told The Sunday Times that presently the virus is completely undetectable in the man’s blood and if it remains that way it will be the first complete cure.

A team assembled from five UK universities is currently conducting experiments on 50 people. “We are exploring the real possibility of curing HIV. This is a huge challenge and it’s still early days but the progress has been remarkable” said Mark Samuels, managing director of the National Institute for Health Research Office for Clinical Research Infrastructure.

HIV is not that easy to treat because it targets the immune system, joining itself into the DNA of T-cells so that they not only ignore the disease, but turn into viral factories which reproduce the virus. Currently, anti-retroviral therapies can target active T-cells which are infected with HIV but they cannot treat dormant T-cells. This means that patient’s body continues to produce the virus.

The new therapy works in two stages. Firstly, a vaccine helps the body recognise the HIV-infected cells so it can clear them out. Secondly, a new drug called Vorinostat activates the dormant T-cells so they can be spotted by the immune system.

“This therapy is specifically designed to clear the body of all HIV viruses, including dormant ones,” Professor Sarah Fidler, a consultant physician at Imperial College London, told the Times.

Fiddler told times “We will continue with medical tests for the next five years and at the moment we are not recommending stopping Art but in the future, depending on the test results we may explore this.”

source: indiatimes

Meet the Woman Who Cared for Hundreds of Abandoned Gay Men Dying of AIDS

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We Cannot Continue to Overlook ‘High-Functioning’ Depression

digital painting of sketched beautiful girl in hat, acrylic on canvas texture

By Amanda Leventhal

I first saw a psychiatrist for my anxiety and depression as a junior in high school. During her evaluation, she asked about my classes and grades. I told her that I had a 4.0 GPA and had filled my schedule with Pre-AP and AP classes. A puzzled look crossed her face. She asked about my involvement in extracurricular activities. As I rattled off the long list of groups and organizations I was a part of, her frown creased further.

Finally, she set down her pen and looked at me, saying something along the lines of, “You seem to be pretty high-functioning, but your anxiety and depression seem pretty severe. Actually, it’s teens like you who scare me a lot.”

Now I was confused. What was scary about my condition? From the outside, I was functioning like a perfectly “normal” teenager. In fact, I was somewhat of an overachiever. I was working through my mental illnesses and succeeding, so what was the problem?

I left that appointment with a prescription for Lexapro and a question that I would continue to think about for years. The answer didn’t hit me all at once; rather, it came to me every time I heard a suicide story on the news saying, “by all accounts, they were living the perfect life.” It came to me as I crumbled under pressure over and over again, doing the bare minimum I could to still meet my definition of success. It came to me as I began to share my story and my illness with others, and I was met with reactions of “I had no idea” and “I never would have known.”

It’s easy to put depression into a box of symptoms, and though we as a society are constantly told mental illness comes in all shapes and sizes, we are stuck with a mental health stock image in our heads that many people don’t match. When we see depression and anxiety in adolescents, we see teens struggling to get by in their day-to-day lives. We see grades dropping. We see involvement replaced by isolation. People slip through the cracks.

We don’t see the student with the 4.0 GPA. We don’t see the student who’s active in choir and theater or a member of the National Honor Society. We don’t see the student who takes on leadership roles in a religious youth group. No matter how many times we are reminded that mental illness doesn’t discriminate, we revert back to a narrow idea of how it should manifest, and that is dangerous.

Recognizing that danger is what helped me find the answer to my question. Watching person after person, myself included, slip under the radar of the “depression detector” made me realize where that fear comes from. My psychiatrist knew the list of symptoms, and she knew I didn’t necessarily fit them. She understood it was the reason that, though my struggles with mental illness began at age 12, I didn’t come to see her until I was 16. Four years is a long time to deal with mental illness alone, and secondary school is a dangerous time to deal with it.

If we keep allowing our perception of what mental illness looks like to dictate how we go about recognizing and treating it, we will continue to overlook those who don’t fit the mold. We cannot keep forgetting that there are people out there who, though they may not be able to check off every symptom on the list, are heavily and negatively affected by their mental illness. If we forget, we allow their struggle to continue unnoticed, and that is pretty scary.

Source: TheMighty.com

Former Horse Jumper With Parkinson’s Disease Gets Her Wish to Ride One More Time

An 87-year-old former horse jumper with Parkinson’s disease got her wish to ride again.

Nelly Jacobs, of the Netherlands, was diagnosed with the debilitating disease more than three decades ago, forcing her to give up riding. Now she spends most her days in a wheelchair in hernursing home.

But, thanks to Hidden Desires, a project of two charities called The Care Group and the Green Cross, Jacobs was granted her wish to ride a horse again at a nearby riding school.

“Horses have always been important to her; ever since she was a child, until her Parkinson’s got the best of her,” Jacobs’ son Jan said in a now viral Facebook video.

He added, “Her uncle Fons put her on a horse when she was 9. Later on, she rode several horses and she even made a decent jumper.”

Jan accompanied his mother to the riding school where she was able to meet several horses in a stable and even smell the hay, an aroma she called “delicious,” before riding.

“This is a very emotional day for her,” her son said. “So she won’t be able to speak, but I can tell she loves every second of seeing the horses, smelling the stables,” her son said. “These things matter to her because she must have missed it at the nursing home.”

After her meet-and-greet, Jacobs was brought to an enclosed arena. She was then gently lifted onto the backs of two horses, who rode side-by-side.

Jacobs called the experience “wonderful” as a broad smile appeared on her face.

“Lots of memories are coming back to her,” her son said in the video. “Her whole life revolved around horses, so through this experience, she’s able to feel the joy again and reminisce about things from her past.”

Source: Yahoo.com

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A Rett Mothers Story

A Rett Mother’s Story

Lara and Abbey

 

Rett-Syndrome-AbbeyI was young and unmarried when I discovered that I was pregnant.  The father of my baby tried to force me to have an abortion, but I refused.  He even went so far as to offer to pay me.  When I still refused, he toldme that he wanted nothing to do with “it” and that our child would suffer for the rest of our lives if I went ahead with the pregnancy.

My pregnancy was perfect.  I drank no alcohol and I ate well.  The birth itself was rather traumatic, but that is another story.  Abbey was a beautifully behaved baby though a bit slow to meet every milestone.

Just before her first birthday I took her to the doctor because she was ill.  At that point the doctor suggested that Abby might be mentally retarded.  An MRI was done but came back clear.

An appointment was made with a pediatric neurologist, who said that there was shrinkage on the left side of her brain caused by lack of oxygen during the birth process. He diagnosed Abbey with Cerebral Palsy.

There were so many tests.  It seemed as if there were millions of blood tests and urine tests as well as many others.  I was confused and frightened.  After her last vaccination she developed a high fever, but when I called the doctor, she just said I was over-exagerating.  Naturally, I went off to my local GP who provided antibiotics.

From the GP’s rooms, I went over to a nearby Spar shop.  Abbey had been sitting in the trolley eating a chicken nugget when suddenly her eyes rolled back and she simply stopped moving.  I checked for food in her mouth in case she was choking, but there was nothing.  Her breathing stopped and she turned blue all over and became ice cold.

I was absolutely terrified and started screaming for my mother (although she wasn’t there), at the same time I ripped Abby out of the shopping trolley and ran back to the GP’s rooms.  By this time she had started breathing again.  Her pediatrician was called and Abbey was rushed to the emergency rooms at the hospital where she had another massive convulsion even as they worked to bring her temperature down.

A lumber puncture was done to check for meningitus.  My poor child screamed blue murder and they would not let me near her.  Fortunately, she tested clear for meningitus.

As time went on, Abbey began to regress.  She stopped clapping and waving her hands in the normal way; she lost the ability to feed herself; she stopped saying the few words she had learned.  Rett-Syndrome-Abbey3

Then, just before her second birthday the strange repetitive hand movements began and which now prevents her from being able to hold anything and from using her hands effectively.

The Pediatric Neurologist told me it was just a bad habit and I needed to break it.  I was not happy with this so I found another Pediatric Neurologist, Dr Crutchley, but had to wait 3 months for my first appointment.  While I waited still more tests were done on Abbey.  I also took her to a pediatrician who said he suspected Angelman syndrome plus autism.

It was also during this time that my mother happened to go to the hairdresser and during a discussion, another client mentioned that her daughter also had the same hand movements and that the child had been diagnosed as having Rett Syndrome.

As soon as my mother told me, I Googled Rett Syndrome.  I was shattered!  Completely heartbroken.  The symptoms matched my daughters behavior to a T.  I felt as if she was dying.  I cried day in and day out and was constantly being sent home from work.

My wonderful dreams for my daughter vanished and left an empty space.  My little ballet dancer would never dance for her mom.  My precious little girl would never tell her mommy she loves her.  Words I so long to hear, will always be left unsaid.

When I finally saw Dr Crutchley for the first time, I decided not to mention Rett Syndrome.  He examined Abbey for about 2 hours, before sitting me down and telling me he believed she had Rett Syndrome.

You should have seen the look on his face when he told me!  I thought he was going to cry.  I told him that I already knew.  I also mentioned that her eye blinking seemed strange to me, which seemed to worry him even more.

Abbey was sent for another EEG  which came back showing that she had multi-focal epilepsy.  Blood tests were performed to check for Rett Syndrome.  It was three long months before the final result was in and Abbey was confirmed as having Rett Syndrome.

My daughter was now three years old.  She seemed not to know that I even existed.  When I got home from work, she would not even look at me, let alone get excited.  Abbey was in her own world where nothing around her existed.  She would simply sit and stare and laugh to herself.

I sent Abbey to a play group and all of a sudden she changed.  It seems that she needed more stimulation than I was able to give her.  Now she can’t wait to see me and I get wonderful hugs and kisses.  She cries when I leave the room.  Abbey puts up her arms so that I will pick her up and when she is ill, she only wants her mommy.

She has become so aware and different.  Almost as if she has been given permission to see the world around her.  She notices, watches and understands everything now.  Nothing seems to get passed her. She watches the leaves blowing and the birds in the trees. When we go somewhere new she looks around and hops about wanting to explore.

I love her just as she is right now.  I don’t want to change her at all.

We have been through so much together and it has all worked to strengthen me.  I take Rett Syndrome day by day.  I no longer look back to what might have been or ahead to what might still be.  I have settled in my heart that this is who Abbey is and how she will be forever.

Before, I could not go into a park and see other children.  It hurt too much.  Now I can go anywhere with her and feel so proud to have her with me.  I love telling people about her and about Rett Syndrome.
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Abbey’s problems and Rett Syndrome have woken me up big time and with the strength of God, I now have the courage and strength to deal with it.

Sadly, her father still throws that fact that I could have avoided this, if only I had aborted her when he wanted me to.

This was my choice!  I would not change anything if I had to do it again.  I am so glad I had Abbey.  She is my everything!

 

source:  rettsyndromesouthafrica

Doctor Says Clinton Collapse Characteristic Of Parkinson’s – Not Just Pneumonia

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CNN is reporting, in what we would expect to be a public relations measure on behalf of their candidate, that Hillary Clinton has pneumonia and that was the cause of her collapsing as she left the 9/11 ceremony early. According to them it was diagnosed on Friday and she’s taking antibiotics.

Clinton emerged from her daughter’s New York City apartment a short while after she went to the saying she felt great. To reassure the American people, Clinton said, “It’s a beautiful day in New York.” Ever the liar caught in a contradiction, Clinton had previously claimed to have been overheated at the morning event. Now, later in the day, when things have warmed up, the weather has turned beautiful.

Her doctor, Dr. Lisa Bardack issued a cover story saying that Clinton was diagnosed on Friday as having pneumonia, and “was put on antibiotics, and advised to rest and modify her schedule.” After attending to Clinton at her Chappaqua home on Sunday, Bardack said, “While at this morning’s event, she became overheated and dehydrated. I have just examined her and she is now re-hydrated and recovering nicely.”

This doctor is an unethical liar. She first stated that Clinton was fit and in excellent physical condition for the office of president, citing only allergies as an ongoing condition. Clearly she has a chronic condition, likely Parkinson’s Disease, and the doctor has committed malpractice in lying on the certification letter.

She’s also lying now. Healthy women don’t pass out like Clinton did, even from a mild case of pneumonia. They don’t have coughing fits and they don’t bob their head uncontrollably and have other involuntary body movements.

It may be true that she has pneumonia. Dr. Ted Noel notes that Aspiration Pneumonia is the leading cause of death for Parkinson’s Disease patients. Aspiration Pneumonia is a lung infection that develops after you aspirate (inhale) food, liquid, or vomit into your lungs.  Far from excluding a more serious condition, the admission only serves to reinforce his suspicions, which are detailed in the video below.

Mrs. Clinton needs to do what is for her unimaginable, be honest. She needs to tell the truth about her health. She has no choice. It is inevitable. She can’t hide any longer. As her fellow mobsters of the past would say, the jig is up.

Source: Rickwells.us

Non-verbal Rett Syndrome sufferer is ‘teaching herself to spell’, says expert

A world-renowned Rett Syndrome communication specialist visited Aylesbury to meet a little girl with the condition – and revealed that she is teaching herself how to spell.

Susan Norwell is based in the US and works with those who are non-verbal – in particular those with Rett Syndrome.

She flew over to meet five-year-old Esmé Davison-Hoult, who was diagnosed with the condition when she was three years old.

The expert spent three days with Esmé and her family in Wendover after the Davison-Hoults raised the cash through fundraising with Esmé’s Allstars, the charity they set up to help their daughter.

During her visit, she spent time with the youngster both at home and at school and revealed to parents Anthony and Abigail that their daughter can recognise words and is teaching herself to spell, despite no structured teaching.

On the Esmé’s Allstars Facebook page, the family said: “She worked Esmé hard getting some fantastic results. It turns out Esmé recognises words in the written form, is starting to spell out short words and has even more to say than we could have imagined.

“She asked for food, for a drink, told us she gets frustrated and sad, told us what she liked about a book, told us the work Susan was asking her to do was too hard (but carried on and did it anyway!).

“It was as exciting and productive an experience as we could have hoped for and just reinforces what a smart cookie she is.

“We are so grateful for all the support we have had to be able to achieve so much in such a short time but we need to do more. So please help us carry on our efforts and if possible support us with our fundraising.

“Esmé has so much to tell us and we are so excited by the possibilities the future holds for her.”

Esm� Davison-Hoult, four, with mum Abigail and brother Patrick, two, at home in Wendover

Esm Davison-Hoult, four, with mum Abigail and brother Patrick, two, at home in Wendover

The condition – which is the result of a genetic mutation and affects around one in every 12,000 girls – can affect sufferers in many different ways.

Esmé, who lives in Wendover with dad Anthony, mum Abigail and brother Patrick, two, spent three days with Susan and Abigail says it was ‘incredible’.

The youngster marked her fifth birthday on Saturday with a fun party with her friends, but her family say it’s always ‘a bittersweet time’.

In a post to the Esmé’s Allstar’s Facebook page, they said: “However, despite the hard times, this year we feel stronger than this time last year.

“We are immeasurably proud of Esmé and the girl that she is growing into. She shows us time and time again how clever she is, how brilliant her sense of humour and social skills are, and we have no doubt she will go on to achieve great things.”

Esmé attends a special school in Aylesbury and her mum says she is doing ‘really well’.

Mrs Davison-Hoult said: “I’m so proud of her for what she can do. She went for a walk at school the other day, and when I picked her up the teacher told me they had been on a walk for nearly a mile.

“That doesn’t seem like much to some people, but for Esmé it’s amazing.”

Esmé has taken up horse riding lessons with Riding For The Disabled in Weston Turville, which mum says she ‘loves’ and it has the added effect of improving her core strength.

As well as juggling family life, the Davison-Hoults are throwing everything into ensuring there is an active programme of fundraising events throughout the year.

The family successfully hit their fundraising target of £15,000 last year which enabled them to purchase a Tobii Eye Gaze computer which Esmé can use both at home and at school to help her communicate.

But Mrs Davison-Hoult says they need to keep fundraising to secure a future for their daughter.

She said: “With the Eye Gaze it was a very distinct thing, we were giving a little girl a voice.

“The next big thing we want to do is to adjust our house. We will need to adapt it for Esmé’s needs by widening the doorways for a wheelchair, and the bathroom and her bedroom will need to be modified too.”

The next one is a curry night at The Raj in Wendover on Tuesday, June 9. Tickets are £20, can be purchased through Facebook here and include starter, main, side and bread, plus the chance to win a Jimmy Choo handbag in the raffle.

Source: Tracykiss.com