‘No Wheelchair Doesn’t Mean He’s Not Disabled’- Term life

Mum Hannah Garlick hits back at people who abuse her for parking in disabled spaces

A YOUNG Sydney mum has started on a mission to teach people that not every person with a disability is in a wheelchair after constant harrassment for parking in disabled parking spaces.Hannah Garlick’s little boy Caleb suffers a range of debilitating illnesses including eosinophilic esophagitis, an allergic inflammatory condition, laryngomalacia and severe eczema which requires daily wet bandages to be applied to much of his body.

While dropping her daughter to school last week, Ms Garlick parked in the disabled parking spot on the grounds as she needed to get back home quickly with Caleb to meet the nurse for his treatment.

After getting out of her car, which was clearly displaying a disabled parking sticker, she was approached by someone who told her she wasn’t allowed to park there.

In an emotional video posted on Facebook, Ms Garlick told of how the confrontation was just one of many she has had while trying to look after her sick child.

She vowed to start a campaign to educate people that not everyone who has a disability always has physical signs that prove it.


When Caleb is without his bandages, Ms Garlick often gets harassed for parking in disabled parking zones. Picture: Facebook

The young mum has created a sign she hopes will educate people on the fact not everyone who has a disability is in a wheelchair.

“Just because he’s not in a wheelchair, doesn’t mean he’s not disabled,” she said.

“Just because he’s a little boy and some days he looks amazing and looks normal, does not mean there’s no disability there.”

When Caleb has been bandaged – sometimes covering his entire body – people are less likely to confront Ms Garlick, but when he has them off, people are much less kind.

“That is an everyday thing. What annoys me the most is when he isn’t wet bandaged I get hammered, absolutely hammered,” she said.

“The amount of people that leave notes on my car and it is so hard because people can barely understand what you go through, let alone (having to) get hammered every day.”

It is a similar story to that suffered by double transplant recipient Lauren Rowe who this week shared a note that had been left on her car blasting her for parking in a disabled parking space at Macarthur Square.


Ms Garlick said she is hoping to change perceptions in Australia with her campaign.

“To the 2 young women who parked here and walked off – your lack of consideration is acknowledged. Well done,” the note read.

Ms Rowe, who had the double lung transplant at 19 but only has 30% capacity after her body began to reject her body, hit back at the woman who left the note and pleaded with people to understand that not all disabilities were visible.

“Unfortunately, this is not the first judgment made against me from using a disabled sticker,” Lauren wrote in a post on her Facebook page that has been liked more than 2000 times and drawn outrage from hundreds of commenters.


Double transplant recipient Lauren Rowe has also spoken out about the problem.

In an effort to change the mindset of others who may be ill-informed about what type of people can use disabled parking spots, Ms Garlick has designed a sign that highlights that not everyone fits into the same single category.

“Not all disabilities look like this,” the sign reads next to a picture of a wheelchair. “Some disabilities look like this..”

Ms Garlick, who says in the video she is looking to change perceptions in Australia, hopes people will see the sign and open their minds before leaving nasty notes or saying insulting things.

Delicious Low Carb Cabbage Casserole- Best weight loss program

New Recipe! Weight Watchers 4 Point Delicious Cabbage Casserole

1 Pound 93% Lean Ground Turkey
1 Container of Fat Free Ricotta Cheese 15 oz
2 Cans of Diced Tomatoes 14.5 oz (one drained, one undrained)
1 Can Tomato Sauce (15 oz)
1/2 Cup Diced Onion
1 Tablespoon Garlic Powder
2 to 3 Teaspoons of Salt
Pepper to taste
1 Medium Head of Cabbage (Diced)

Preheat oven to 350 Degrees
Brown the ground turkey and then throw in the 1/2 Cup of onions
Simmer for 3 to 4 minutes
Add Diced Tomatoes, Entire can of tomato Sauce and seasonings
Let Simmer for about 15 minutes
While that is simmering Spray a 3 quart baking dish with Pam or Canola Spray
Add 4 Cups of Cabbage on the bottom of the pan
Now take your meat sauce and put half of it on top of the cabbage
Add 3 to 4 Cups of Cabbage and then top with the remaining meat sauce
Take the entire container of Ricotta and spread evenly over the entire dish
Cover with aluminum foil and Bake for 40 to 45 minutes
Remove aluminum foil and bake for an additional 15 to 20 minutes (until the top is lightly brown)
Cut into 8 Slices! Each huge slice is only 4 Points.

Source

10 Rules for Parenting a Child with Sensory Processing Disorder- Advanced mold remediation

Every year, thousands of young children are diagnosed with disorders that make it difficult for them to absorb the external world. Parents of sensory kids—like those with sensory processing disorder, AD/HD, autism, bipolar disorder, and OCD—often feel frustrated and overwhelmed, creating stress in everyday life for the whole family.

 

sensory processing disorder in children

I know a little bit about this myself. I’m the parent of a sensory child, as well as the owner of a business founded to help these kids and their families function in the world. So I know firsthand the struggles that parents face in trying to bring out the best in their rigid, anxious, or distracted sensory children. In my work with my own family, as well as with the countless families I’ve worked with in my company, Systems for Sensory Kids, I’ve come up with my own rules for parenting a sensory child—some guidelines to keep in mind when things get tough. While they are especially applicable for parents of sensory children, in reality ALL of us can benefit from keeping these truisms front of mind when we face tough situations with our kids.

 

Your job is not to compare yourself to other parents around you, but to figure out what works for your own family. Here are a few new parenting rules to help get you started.

Your child’s disorder is not a reflection on you or your parenting. Looking at things for how they really are and letting go of the why or how it happened can get us to a neutral, open place. You will need to gain a level of confidence that these new parenting rules are right for your child. The reality is that parents of typical children or members of your own family will question your approach. They are usually coming from a place of wanting to help, but have no frame of reference or experience with sensory children. Stick to your guns! Only you know what is best for your sensory child and your family.

 

Let go of guilt and anger. When you are in a place of blame, guilt, or anger, you are making your sensory child’s experience about you, and this takes away your power to advocate for them effectively.

 

Value the gift of the experience. Get in a habit of sitting down and writing out a gratitude list of all the wonderful things you have learned and experienced as a result of being the parent of a sensory child.

 

Initially, parenting a sensory child is a counter intuitive process. What might work when parenting most typical kids usually will not work in the same way for sensory kids. It takes more conscious thought and preparation for daily activities to parent a sensory child. If you can be mindful of this one idea, you will be able to adjust and adapt your plans to the daily situations that might be a challenge.

 

Celebrate your child’s strengths. Have a solid understanding of your sensory child’s strengths. Write out a list of all your child’s great characteristics. Sensory kids are special and among some of the most successful adults in the world. You are going to run into many people who won’t understand or appreciate what they bring to the table—make sure you do!

Parenting a sensory child is a marathon, not a race. Parenting is a journey and with a sensory child, the journey tends to have many twists and turns. Focus on the long-term objectives and then create the steps needed to get there.

No sensory solution works forever. Frequent amendments will be needed to support your growing and ever-changing sensory child. All kids grow and change, and these changes can be more exaggerated for sensory kids. When you understand how to tap into structure, routines, and visual aids, you will be able to find solutions to the changing landscape you will face over time with your sensory child.

 

Embrace when you do it all wrong. The bottom line is that you can learn more about how to better support your sensory child when something goes all wrong as opposed to the hundred times you do it right. Embrace the lessons in the “wrong” experiences.

 

Be guided by love and understanding. Our sensory kids just want to feel safe, loved, and understood. They are great kids who have a hard time learning the rules of life in the traditional way. They need and want to have times every day when they are in an environment that they understand and that supports their way of seeing the world. You can do this for them at home.

 

Pass it on. One of the best long-term gifts we can give sensory children is to teach them the tools. If you start sensory organizing at an early age, your sensory child will have years of practice, trial and error, and examples of real success. The goal is for this to be a way of life for them so when they are in high school and feeling overwhelmed, they stop and say, “What’s my plan to handle this or get this done?” That is the definition of self-reliance (and successful parenting).

Source

The facts about Addison’s Disease Missed by Mostly People- Term life

What is adrenal insufficiency?

Adrenal insufficiency is an endocrine-or hormonal-disorder that occurs when the adrenal glands do not produce enough of certain hormones. The adrenal glands are located just above the kidneys. Adrenal insufficiency can be primary or secondary.

Primary adrenal insufficiency, also called Addison’s disease, occurs when the adrenal glands are damaged and cannot produce enough of the hormone cortisol and often the hormone aldosterone. Addison’s disease affects one to four of every 100,000 people, in all age groups and both sexes.1

Secondary adrenal insufficiency occurs when the pituitary gland-a bean-sized organ in the brain-fails to produce enough adrenocorticotropin (ACTH), a hormone that stimulates the adrenal glands to produce cortisol. If ACTH output is too low, cortisol production drops. Eventually, the adrenal glands can shrink due to lack of ACTH stimulation. Secondary adrenal insufficiency is much more common than Addison’s disease.

1Munver R, Volfson IA. Adrenal insufficiency: diagnosis and management. Current Urology Reports. 2006;7:80’€“85.

What do adrenal hormones do?

cortisol

Cortisol belongs to a class of hormones called glucocorticoids, which affect almost every organ and tissue in the body. Cortisol’s most important job is to help the body respond to stress. Among its many vital tasks, cortisol helps

  • maintain blood pressure and cardiovascular function
  • slow the immune system’s inflammatory response
  • maintain levels of glucose-a form of sugar used for energy-in the blood
  • regulate the metabolism of proteins, carbohydrates, and fats

The amount of cortisol produced by the adrenals is precisely balanced. Like many other hormones, cortisol is regulated by the brain’s hypothalamus and the pituitary gland. First, the hypothalamus releases a “trigger” hormone called corticotropin-releasing hormone (CRH) that signals the pituitary gland. The pituitary responds by sending out ACTH, which in turn stimulates the adrenal glands. The adrenal glands respond by producing cortisol. Completing the cycle, cortisol then signals back to both the pituitary and hypothalamus to decrease these trigger hormones.

Drawing of the brain and adrenal glands within an outline of a male torso. The hypothalamus, pituitary gland, adrenal glands, and kidneys are labeled. Arrows show the effect of CRH on ACTH and the effect of ACTH on cortisol.
The hypothalamus sends CRH to the pituitary, which responds by sending out ACTH. ACTH then causes the adrenals to release cortisol into the bloodstream.

What are the symptoms of adrenal insufficiency?

The symptoms of adrenal insufficiency usually begin gradually. The most common symptoms are

  • chronic, worsening fatigue
  • muscle weakness
  • loss of appetite
  • weight loss

Other symptoms can include

  • nausea
  • vomiting
  • diarrhea
  • low blood pressure that falls further when standing, causing dizziness or fainting
  • irritability and depression
  • a craving for salty foods due to salt loss
  • hypoglycemia, or low blood glucose
  • headache
  • sweating
  • in women, irregular or absent menstrual periods

Hyperpigmentation, or darkening of the skin, can occur in Addison’s disease but not in secondary adrenal insufficiency. This darkening is most visible on scars; skin folds; pressure points such as the elbows, knees, knuckles, and toes; lips; and mucous membranes such as the lining of the cheek.

Because the symptoms progress slowly, they are often ignored until a stressful event like an illness or accident causes them to worsen. Sudden, severe worsening of symptoms is called an Addisonian crisis, or acute adrenal insufficiency. In most cases, symptoms of adrenal insufficiency become serious enough that people seek medical treatment before a crisis occurs. However, sometimes symptoms first appear during an Addisonian crisis.

Symptoms of an Addisonian or “adrenal” crisis include

  • sudden, penetrating pain in the lower back, abdomen, or legs
  • severe vomiting and diarrhea
  • dehydration
  • low blood pressure
  • loss of consciousness

If not treated, an Addisonian crisis can be fatal.

What causes Addison’s disease?

autoimmune disorders

The gradual destruction of the adrenal cortex, the outer layer of the adrenal glands, by the body’s immune system causes up to 80 percent of Addison’s disease cases.2 In autoimmune disorders, the immune system makes antibodies that attack the body’€™s own tissues or organs and slowly destroy them.

Adrenal insufficiency occurs when at least 90 percent of the adrenal cortex has been destroyed. As a result, often both cortisol and aldosterone are lacking. Sometimes only the adrenal glands are affected. Sometimes other endocrine glands are affected as well, as in polyendocrine deficiency syndrome.

Polyendocrine deficiency syndrome is classified into two separate forms, type 1 and type 2. Type 1 is inherited and occurs in children. In addition to adrenal insufficiency, these children may have

  • underactive parathyroid glands, which produce a hormone that regulates calcium and phosphorus balance in the body
  • slow sexual development
  • pernicious anemia, a severe type of anemia
  • chronic candida infections, a type of fungal infection
  • chronic active hepatitis, a liver disease

Type 2, sometimes called Schmidt’s syndrome, usually affects young adults and may include

  • an underactive thyroid gland, which produces hormones that regulate metabolism
  • slow sexual development
  • diabetes
  • vitiligo, a loss of pigment on areas of the skin

Scientists think type 2 polyendocrine deficiency syndrome is also inherited because often more than one family member has one or more endocrine deficiencies.

2Martorell PM, Roep BO, Smit JWA. Autoimmunity in Addison’€™s disease. The Netherlands Journal of Medicine. 2002;60(7):269-275

tuberculosis

Tuberculosis (TB), an infection that can destroy the adrenal glands, accounts for less than 20 percent of cases of Addison’s disease in developed countries.3 When adrenal insufficiency was first identified by Dr. Thomas Addison in 1849, TB was the most common cause of the disease. As TB treatment improved, the incidence of adrenal insufficiency due to TB of the adrenal glands greatly decreased.

3Munver R, Volfson IA. Adrenal insufficiency: diagnosis and management.Current Urology Reports. 2006;7:80-85.

other causes

Less common causes of Addison’s disease are

  • chronic infection, mainly fungal infections
  • cancer cells spreading from other parts of the body to the adrenal glands
  • amyloidosis, a disease that causes abnormal protein buildup in, and damage to, various organs
  • surgical removal of the adrenal glands
  • AIDS-associated infections
  • bleeding into the adrenal glands
  • genetic defects including abnormal adrenal gland development, an inability of the adrenal gland to respond to ACTH, or a defect in adrenal hormone production

What causes secondary adrenal insufficiency?

Secondary adrenal insufficiency can be traced to a lack of ACTH. Without ACTH to stimulate the adrenal glands, the adrenals’ production of cortisol drops. Aldosterone production is not usually affected.

A temporary form of secondary adrenal insufficiency may occur when a person who has been taking a synthetic glucocorticoid hormone such as prednisone for a long time stops taking the medication, either abruptly or gradually. Glucocorticoid hormones, which are often used to treat inflammatory illnesses such as rheumatoid arthritis, asthma, and ulcerative colitis, block the release of both CRH and ACTH. As a result, the adrenals may begin to atrophy or shrink €”from lack of ACTH stimulation and then fail to secrete sufficient levels of cortisol.

A person who stops taking a synthetic glucocorticoid hormone may have enough ACTH to function when healthy. However, when a person is under the stress of an illness, accident, or surgery, the person’s body may not have enough ACTH to stimulate the adrenal glands to produce cortisol.

Another cause of secondary adrenal insufficiency is surgical removal of the noncancerous, ACTH-producing tumors of the pituitary gland that cause Cushing’s disease. Cushing’s disease is another disorder that leads to excess cortisol in the body. In this case, the source of ACTH is suddenly removed and replacement hormone must be taken until normal ACTH and cortisol production resumes.

Less commonly, adrenal insufficiency occurs when the pituitary gland either decreases in size or stops producing ACTH. These events can result from

  • tumors or infections of the area
  • loss of blood flow to the pituitary
  • radiation for the treatment of pituitary tumors
  • surgical removal of parts of the hypothalamus
  • surgical removal of the pituitary gland

How is adrenal insufficiency diagnosed?

In its early stages, adrenal insufficiency can be difficult to diagnose. A review of a patient’€™s medical history and symptoms may lead a doctor to suspect Addison’€™s disease.

A diagnosis of adrenal insufficiency is confirmed through laboratory tests. The aim of these tests is first to determine whether levels of cortisol are insufficient and then to establish the cause. Radiologic exams of the adrenal and pituitary glands also are useful in helping to establish the cause.

acth stimulation test

The ACTH stimulation test is the most commonly used test for diagnosing adrenal insufficiency. In this test, blood cortisol, urine cortisol, or both are measured before and after a synthetic form of ACTH is given by injection. The normal response after an ACTH injection is a rise in blood and urine cortisol levels. People with Addison’s disease or long-standing secondary adrenal insufficiency have little or no increase in cortisol levels.

Both low- and high-dose ACTH stimulation tests may be used depending on the suspected cause of adrenal insufficiency. For example, if secondary adrenal insufficiency is mild or of recent onset, the adrenal glands may still respond to ACTH because they have not yet atrophied. Some studies suggest a low dose-1 microgram-may be more effective in detecting secondary adrenal insufficiency because the low dose is still enough to raise cortisol levels in healthy people but not in people with mild or recent secondary adrenal insufficiency.

crh stimulation test

When the response to the ACTH test is abnormal, a CRH stimulation test can help determine the cause of adrenal insufficiency. In this test, synthetic CRH is injected intravenously and blood cortisol is measured before and 30, 60, 90, and 120 minutes after the injection. People with Addison’€™s disease respond by producing high levels of ACTH but no cortisol. People with secondary adrenal insufficiency have absent or delayed ACTH responses. CRH will not stimulate ACTH secretion if the pituitary is damaged, so an absent ACTH response points to the pituitary as the cause. A delayed ACTH response points to the hypothalamus as the cause.

diagnosis during an emergency

In patients suspected of having an Addisonian crisis, health professionals must begin treatment with injections of salt, glucose-containing fluids, and glucocorticoid hormones immediately. Although a reliable diagnosis is not possible during crisis treatment, measurement of blood ACTH and cortisol during the crisis-before glucocorticoids are given-is enough to make a preliminary diagnosis. Low blood sodium, low blood glucose, and high blood potassium are also usually present at the time of an adrenal crisis. Once the crisis is controlled, an ACTH stimulation test can be performed to obtain the specific diagnosis. More complex laboratory tests are sometimes used if the diagnosis remains unclear.

other tests

Once a diagnosis of Addison’s disease is made, radiologic studies such as an x ray or an ultrasound of the abdomen may be taken to see if the adrenals have any signs of calcium deposits. Calcium deposits may indicate bleeding in the adrenal gland or TB, for which a tuberculin skin test also may be used. Blood tests can detect antibodies associated with autoimmune Addison’s disease.

If secondary adrenal insufficiency is diagnosed, doctors may use different imaging tools to reveal the size and shape of the pituitary gland. The most common is the computerized tomography (CT) scan, which produces a series of x-ray pictures giving cross-sectional images. A magnetic resonance imaging (MRI) scan may also be used to produce a three-dimensional image of this region. The function of the pituitary and its ability to produce other hormones also are assessed with blood tests.

How is adrenal insufficiency treated?

Treatment of adrenal insufficiency involves replacing, or substituting, the hormones that the adrenal glands are not making. Cortisol is replaced with a synthetic glucocorticoid such as hydrocortisone, prednisone, or dexamethasone, taken orally once to three times each day, depending on which medication is chosen. If aldosterone is also deficient, it is replaced with oral doses of a mineralocorticoid, called fludrocortisone acetate (Florinef), taken once or twice a day. Doctors usually advise patients receiving aldosterone replacement therapy to increase their salt intake. Because people with secondary adrenal insufficiency normally maintain aldosterone production, they do not require aldosterone replacement therapy. The doses of each medication are adjusted to meet the needs of the individual.

During an Addisonian crisis, low blood pressure, low blood glucose, and high levels of potassium can be life threatening. Standard therapy involves intravenous injections of glucocorticoids and large volumes of intravenous saline solution with dextrose, a type of sugar. This treatment usually brings rapid improvement. When the patient can take fluids and medications by mouth, the amount of glucocorticoids is decreased until a maintenance dose is reached. If aldosterone is deficient, maintenance therapy also includes oral doses of fludrocortisone acetate.

What special problems can occur with adrenal insufficiency?

surgery

Because cortisol is a “stress hormone,” people with chronic adrenal insufficiency who need any type of surgery requiring general anesthesia must be treated with intravenous glucocorticoids and saline. Intravenous treatment begins before surgery and continues until the patient is fully awake after surgery and able to take medication by mouth. The “stress” dosage is adjusted as the patient recovers until the presurgery maintenance dose is reached.

In addition, people who are not currently taking glucocorticoids but who have taken long-term glucocorticoids in the past year should tell their doctor before surgery. These people may have sufficient ACTH for normal events, but they may need intravenous treatment for the stress of surgery.

illness

During illness, oral dosing of glucocorticoid may be adjusted to mimic the normal response of the adrenal glands to this stress on the body. Significant fever or injury may require triple oral dosing. Once recovery from the stress event is achieved, dosing is then returned to maintenance levels. People with adrenal insufficiency should know how to increase medication during such periods of stress. Immediate medical attention is needed if severe infections, vomiting, or diarrhea occur. These conditions can precipitate an Addisonian crisis.

pregnancy

Women with adrenal insufficiency who become pregnant are treated with standard replacement therapy. If nausea and vomiting in early pregnancy interfere with taking medication by mouth, injections of the hormone may be necessary. During delivery, treatment is similar to that of people needing surgery. Following delivery, the dose is gradually tapered and the usual maintenance doses of oral hydrocortisone and fludrocortisone acetate are reached about 10 days after childbirth.

How can someone with adrenal insufficiency prepare for an emergency?

People with adrenal insufficiency should always carry identification stating their condition in case of an emergency. A card or medical alert tag should notify emergency health care providers of the need to inject cortisol if the person is found severely injured or unable to answer questions. The card or tag should also include the name and telephone number of the person’€™s doctor and the name and telephone number of a family member to be notified. The dose of hydrocortisone needed may vary with a person’s age or size. For example, a child younger than 2 years of age can receive 25 milligrams (mg), a child between 2 and 8 years of age can receive 50 mg, and a child older than 8 years should receive the adult dose of 100 mg. When traveling, people with adrenal insufficiency should carry a needle, syringe, and an injectable form of cortisol for emergencies.

Points to Remember

  • Adrenal insufficiency is a disorder that occurs when the adrenal glands do not produce enough of certain hormones.
  • Primary adrenal insufficiency, also called Addison’s disease, occurs when the adrenal glands are damaged and cannot produce enough of the hormone cortisol and often the hormone aldosterone.
  • Secondary adrenal insufficiency occurs when the pituitary gland fails to produce enough adrenocorticotropin (ACTH), a hormone that stimulates the adrenals to produce cortisol. If ACTH output is too low, cortisol production drops.
  • The most common symptoms of adrenal insufficiency are chronic, worsening fatigue; muscle weakness; loss of appetite; and weight loss.
  • Adrenal insufficiency is most often diagnosed through blood or urine tests. Imaging studies such as x rays, ultrasound, computerized tomography (CT), and magnetic resonance imaging (MRI) may also be used.
  • Treatment of adrenal insufficiency involves replacing, or substituting, the hormones that the adrenal glands are not making.
  • People with adrenal insufficiency should always carry identification stating their condition in case of an emergency.

    Source

Crock Pot Low Carb Un-Stuffed Cabbage Roll Soup- Best weight loss program

Crock Pot Un-Stuffed Cabbage Roll Soup is a comforting, hearty, but low carb meal you can make any weeknight!

Are you a cabbage fan?

Even if you aren’t, this recipe might just win you over. One of the most comforting meals on a cold winter’s day made in your crock pot or just simmer on the stove, but either way it’s a hearty meal for the whole family.

cabbage roll soup1 (1 of 1)

Did you make any New Year’s Resolutions?

I don’t. I do make short term goals though and feel with a goal, I make steps each day toward reaching it. A monthly or 3 month or even 6 month goal is far easier to see the end result than a whole year long resolution don’t you agree?

cabbage roll soup5 (1 of 1)

My hubby finally made a decision (well New Year’s resolution!) to remove the carbs from his diet. I have to say after many years waiting for my man to want to make healthier changes in his life I’m pretty excited about this.

Oh don’t misunderstand though, he eats everything I make at home. It’s when he’s not home, he chooses his love of carb and sugar laden foods and goodies.

cabbage roll soup2 (1 of 1)

Getting your love to desire to be healthier has to be their choice and one I haven’t tried to push because in all honestly you just can’t make someone want something they don’t want. Am I right?

But finally after many years he is focusing on the carbs and sugar and feeling much better all ready.

cabbage roll soup6 (1 of 1)

My job is making sure there is plenty of low carb and sugar free foods for him to eat at home that keeps him full and satisfied and this soup is one of those recipes. He had it for lunch every day last week!

The first time I served this was for a New Year’s Day party with over 50 people and it went fast and everyone was asking for the recipe! I’ve made them all wait until today LOL!

cabbage roll soup4 (1 of 1)

If you’ve had a reluctant family member not ready to embrace a sugar free and/or low carb lifestyle, try comforting recipes like this that they won’t even notice is grain free and healthy! They will hopefully realize it’s not that hard to give up processed carbs and sugar.

Here’s to a New Healthy Year Ahead!

  • You can eliminate the cauliflower in the recipe if you prefer. I feel it was a good substitute for rice which is usually in cabbage rolls. I think it thickened it nicely without being very noticeable in texture.
  • The sugar amount you see listed below is naturally made from the tomato sauce used.
  • You could simply use cooked rice in place of the amount of cauliflower I used.
  • I will tell you that not even my hubby noticed cauliflower was in this soup and everyone raved about it!
  • Are you unsure about all the sugar free sweetener options and what is the difference betweensugar free versus low carb? Here’s what you’ll see shared on my blog as well. If you’re all ready a sugar free fan, but need a little guidance in converting some recipes to sugar free, this post about all the differences with stevia plus a conversion chart might help you.
  • And last if you’re ready to start the New Year, but need a little more help and guidance you can check out my Sugar Detox Plan and well as my Cookbook for more helpful recipes.
  • You can subscribe for free by email so you won’t ever miss any recipes I share on social media and just for subscribing you receive a FREE recipe from my Cookbook!
Crock Pot Low Carb Un-Stuffed Cabbage Roll Soup
  • Serves: 9
  • Serving size: 1 1/2 cups
  • Calories: 312
  • Fat: 15.2g
  • Saturated fat: 5g
  • Carbohydrates: 12.6g
  • Sugar: 4.5g
  • Sodium: 554mg
  • Fiber: 2.8g
  • Protein: 31.1g
  • Cholesterol: 84mg
Recipe type: Entree
Prep time: 
Cook time: 
Total time: 
Ingredients
  • 2 tbsp extra virgin olive oil
  • 2 garlic cloves, minced
  • 1/2 cup chopped onioon
  • 1/2 cup chopped shallots
  • 2 pounds ground beef
  • 1 tsp dried parsley
  • 1/2 tsp dried oregano
  • 1 tsp salt
  • 1 tsp pepper
  • 16 ounces marinara sauce
  • 1/2 head cauliflower or 2 cups riced cauliflower
  • 5 cups beef broth, low sodium
  • 1 large cabbage or 8 cups sliced
Instructions
  1. Heat olive oil and garlic on medium high heat.
  2. Add onions and shallots and cook until softened.
  3. Add ground beef and cook until browned and no longer pink.
  4. Add seasonings to beef and marinara sauce.
  5. Add the riced cauliflower to the beef mixture and stir until coated.
  6. Pour the beef into the crock pot.
  7. Pour beef broth into crock pot and add cabbage.
  8. Stir to combine everything.
  9. Cook on high 3 hours or low 6 hours. If you don’t have a crock pot, simply cook ground beef in a dutch oven or heavy soup pot, follow recipe and simmer on low, covered for about an hour until cabbage is tender.
Notes
Net Carbs: 9.8g
You could reduce the carbs more if you have just a cup as a serving instead of suggested serving.
If you choose to use rice instead of cauliflower then I would suggest 1 cup of cooked rice and eliminate the cauliflower.

A CYSTIC fibrosis sufferer whose sister died from the same condition has now been handed a “death sentence” – as a potentially lifesaving drug is to be cut from NHS funding. Term life

‘Death sentence’ girl with cystic fibrosis ‘denied’ life-saving drug ditched by the NHS… months after her sister died from the same disease

Hannah Lindley, from Leeds, has slammed health body NICE after it released a draft guidance recommending the drug Orkambi has its funding cut.

The 20-year-old and her younger sister Iona were both diagnosed with the illness as babies, but Iona’s condition deteriorated far more quickly and she passed away in November.

HANNAH-LINDLEY.
The sisters spent their childhood going to hospital appointments together and having nebuliser treatment each morning before school Mercury Press and Media Ltd

Hannah was horrified in February when NICE advised that as the drug costs £104,000 a year per patient and only provides a ‘modest’ benefit, it is no longer cost-effective.

“When Orkambi was first available, everyone called it a wonder drug and a medical breakthrough. It was making a huge difference to people, so I can’t understand how it’s now in the process of being cut. It’s like being handed a death sentence,” Hannah said.

Hannah’s lung function is at 55 per cent and she says it’s only a matter of time before it gets worse.

As a baby, Hannah experienced jaundice, coughing and wheezing and was unable to gain weight, before finally being diagnosed at 14 months old.

Her younger sister Iona had just been born and was immediately discovered to be a sufferer. Despite the early diagnosis, her condition declined more quickly.

HANNAH-LINDLEY.
Iona and Hannah as children: Hannah’s condition stabilised a little in year one but Iona got more poorly and sadly died in November 2015 Mercury Press and Media Ltd

HANNAH-LINDLEY.
Iona (left) married her childhood sweetheart in May 2013, aged just 16, but sadly lost her battle with her lifelong illness at the end of last year Mercury Press and Media Ltd

The sisters spent their childhood going to hospital appointments together and having nebuliser treatment each morning before school.

Tragically, Iona continued to rapidly lose lung function and died in November last year.

“Me and Iona were inseparable,” Hannah reflected. “Mum always referred to us as twins, as we were so close in age, our facial features and personalities.

“We had the same friends, wore the same clothes and shared everything. When we were well enough we did ballet, tap and piano lessons together.

“Losing her was very hard, even though we were able to prepare for it. We hoped she would get a lung transplant but in May last year she was at 20 per cent function and couldn’t even stand up. She wasn’t strong enough for surgery. Iona died in hospital two days after our mum’s birthday.

HANNAH-LINDLEY.
Hannah has started a petition for the drug orkambi to be made available on the NHS. ‘You can’t put a price on life,’ she said. Mercury Press and Media Ltd

Hannah has started an online campaign in partnership with the Cystic Fibrosis Trust in an attempt to overturn the decision not to prescribe Orkambi.

“I know it costs a heck of a lot of money, but it would prevent other costs. For example in October I had a collapsed bowel and needed an ultrasound, X-ray and MRI scan and I racked up nearly £10,000 then. That would cover over a month of Orkambi,” Hannah said.


“Two of my closest friends with cystic fibrosis died in February, within four days of each other. One was 22 and the other was 23 – people are dying so young and it’s not fair.

“I don’t know how quickly my lungs will decline. I can do everything for myself most of the time, but some days I spend the whole day in my pyjamas as I’m so drained. Lately I’ve not been well, so just getting out of bed is an issue. Making a cup of tea knocks it out of me.”

Hannah has to take so much medication she has a big whiteboard in the utility room to keep track of it.

She needs antibiotics to prevent chest infections, drugs for her liver, more for her stomach and several vitamins and calcium.

HANNAH-LINDLEY.
Hannah, 20, with her various medications including strong pain killers. She has to have her lung function monitored regularly at hospital Mercury Press and Media Ltd

Tests have shown that Orkambi benefits patients by reducing instances where they experience a sudden worsening of symptoms that requires hospitalisation.

Professor Carole Longson, director of the NICE Centre for Health Technology Evaluation, said that the decision was taken very carefully, adding: “Orkambi is a new treatment option and it is disappointing that we are not able to recommend it.

“However our independent committee found that when compared to the current standard of care, the benefit it offered was modest and comes at a considerable cost.

“We have to recognise that the NHS is a finite resource and we can only recommend treatments for routine funding that are both clinically effective and represent good value for money.”

Consul-tees, healthcare professionals and members of the public can still comment on the preliminary recommendations, which are up for public consultation until April 15.

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MS is Actually Lyme Disease- Drug rehab center

The world must be a very scary place for a conspiracy theorist. In their world there is a vast sinister conspiracy that can control entire industries and professions, that supersede governments, and have almost limitless power.  In the mind of a conspiracy theorist the very people who are supposed to help and protect us are instead villains exploiting the public in the most heinous way for their own profit – and not just some individuals, but entire professions.

As experienced as I am examining conspiracy theories I always experience an uncomfortable cognitive dissonance when reading a new conspiracy theory – how can someone actually believe this stuff? Their brain must operate under a different set of algorithms from my own.

Recently I was sent a link to this website claiming, without a hint of self-doubt, that there has been a 100 year conspiracy to lie to the world about multiple sclerosis (MS). Scientists and doctors, they assert, know that MS (and many other neurological diseases, like ALS, Parkinson’s disease, and Alzheimer’s disease) is really caused by a Borrelia infection of the brain – Lyme disease.

The story the author has constructed is a simplistic cardboard caricature with the usual villains and motives. There isn’t even any imagination in this one. Because the website is monetized I always have to suspect that the author is not even sincere – they are just packaging a standard conspiracy theory for their intended audience, in whom they have utter contempt. I have no idea if this is the case, we just cannot assume that those selling conspiracy theories like this always believe their own nonsense.

In any case – what we have here is boringly predictable: Big Pharma wants to maintain their billions in profits from symptom management of disease X so they suppress knowledge of the real cause and cure of disease X and bribe doctors and researchers with funding and kickbacks to toe the corporate line and keep it all hush hush. Yawn. You can substitute any chronic illness for disease X and monetize your own website.

I am always stunned by a couple of things about such claims. First (assuming sincerity) is the utter moral and intellectual arrogance of the conspiracy theorist. They have to believe that they are better able to understand and interpret the scientific research than legions of experts in multiple related fields. Either that or that the entire scientific community is corrupt to the core.  In either case they believe their powers of perception are so keen they can see the conspiracy that others do not. Further, they casually slander entire professions with the most vile of accusations while maintaining their own moral superiority.

It’s a very black and white world with bright lines of demarcation between right and wrong and the conspiracy theorist as the white-hatted crusader.

For the record, MS is not caused by Lyme disease. MS has existed for a much longer time – Borrelia burgdorferi arose in the 1970s and probably existed for longer than that before it broke into the human population. It is possible for MS patients to become infected with Lyme and this does tend to worsen the course of their disease, but it is not a cause of MS.

After Lyme was discovered it was investigated to see if it was the cause of known neurological diseases, including MS. This was almost 40 years ago and much less was known about MS and the extent of Lyme pathology at that time. The research showed, however:

Clinical, neuropathologic, laboratory and epidemiologic features indicate clearly that tertiary Lyme borreliosis of the CNS is a distinct entity and there is no etiologic association with multiple sclerosis.

Every line of medical evidence points to the conclusion that MS and CNS Lyme disease are different diseases.

Think about how prescient “Big Pharma” must be in order to engineer the conspiracy being claimed. They must have recognized very early on that this new discovery of Lyme disease was actually the cause of MS and started manipulating the world’s medical research to keep that from ever coming out. I wonder how they figured this out before the very researchers they are supposed to control? Once you start asking these questions the house of cards starts utterly collapses – unless, of course, you are a conspiracy theorist. If you are, you just broaden the conspiracy, involved more people and institutions, and give the conspirators more and more reach and power until they are secretly controlling the whole world.

One section of the conspiracy article caught my attention as an excellent example of how conspiracy theorists perceive sinister motives in benign places. They quote from a neuroradiology lecture on a Dutch website (the internet is excellent for data mining and cherry picking). They characterize the quote this way:

What we found is deeply worrying. The radiologists are instructed in ominous, derogatory language never to disagree with the “suspicion of MS”. So when the doctor says:  “I think it should be MS”, the radiologist should just shut up and agree, even if he disagrees and thinks it’s Lyme disease.

This is what the text actually says:

If a patient is clinically suspected of having MS and the MR-images support that diagnosis, then you should not consider the possibility of Lyme disease and neuro-SLE in the differential diagnosis, because they have such a low prevalence.

There must be other ways to impress your colleagues. These diagnoses are only worth mentioning if there are clinical findings that support these diagnoses.

Yes – very ominous and derogatory.  This is all, actually, standard fare. Radiologists are not clinicians. They are not in the business of making diagnoses, but in interpreting radiographic studies. Findings on MRI scan and other such studies are rarely specific enough to establish a diagnosis by themselves (although this is changing as MR technology progresses, but that’s another story). Radiology findings need to be put into clinical context, and making a diagnosis is often a collaboration between the radiologist and the clinician.

The MRI findings of MS, Lyme disease, small vessel disease, and various causes of brain inflammation can all look similar on MRI scan – white matter demyelination. So when this is seen on the MRI scan the radiologist will typically give the standard differential of white matter demyelination in the interpretation – “This can represent, in the proper clinical setting, multiple sclerosis, Lyme disease, small vessel ischemic disease, and other causes of demyelination.” I can’t tell you how many times I have read some variation of that in the impression of an MRI scan.

This can be a bit confusing to the inexperienced and scary to patients who increasingly get direct access to their lab results. The radiologists mention everything, and it’s the clinicians job to put it into context.

Further – while MS and Lyme look similar on MRI scans they are not identical. MS often has a distinct distribution of lesions. There are “classic” MS features on MRI scan, although they are not always present. Radiologists, however, tend to cast a broad net, naming everything and letting the treating physician sort it out.

What this lecturer is saying is that radiologists should not just throw in rare diseases in the differential diagnosis on their reading if there is no clinical reason to suspect such illnesses. This is a reasonable suggestion. This was on a Dutch website, so I suspect the lecturer is not practicing in New England. Here radiologists always mention Lyme disease in the differential.  There is nothing sinister or ominous about this recommendation – it simply refers to the relationship between radiological and clinical findings in diagnosing a patient, and the proper role of the radiologist.

I don’t expect a non-expert to understand these nuances, but that is the point. The conspiracy theorist is profoundly naive about how medicine actually operates, and enthusiastically fills the void of their ignorance with sinister assumptions.

While this is all standard grand conspiracy nonsense, such accusations always take on a different dimension when they apply to people you know personally. I do not treat or research MS, but I know many people who do, including colleagues with whom I work. The notion that they are involved in any kind of cover up like this is absurd. These are academics trying to understand the underlying immunological basis of MS, improve our treatments, and help their patients.

To casually slander them as this conspiracy theorist has is vile and reprehensible. Further they are encouraging patients to distrust their doctors and the medical profession, and to forgo effective treatment for a conspiracy fantasy.

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Early Warning Symptoms of Diverticulitis Missed by Mostly People- Drug rehab center

Symptoms of diverticular disease and diverticulitis include abdominal pain, bloating and a change in normal bowel habits.

Diverticulosis

If diverticula have been discovered during a camera test for another reason (colonoscopy) or during a CT scan, you may be worried about what this means.

However, if you have never had abdominal pain or bouts of diarrhoea, there is a 70-80% chance that you will never have any symptoms from them.

Diverticula are extremely common over the age of 70 and they do not increase your risk of cancer. It’s thought that a high-fibre diet is likely to reduce the risk of any symptoms developing.

Diverticular disease

The most common symptom of diverticular disease is intermittent (stop-start) pain in your lower abdomen (stomach), usually in the lower left-hand side.

The pain is often worse when you are eating, or shortly afterwards. Passing stools and breaking wind (flatulence) may help relieve the pain.

Other long-term symptoms of diverticular disease include:

  • a change in your normal bowel habits, such as constipation ordiarrhoea, or episodes of constipation that are followed by diarrhoea – a classic pattern is multiple trips to the toilet in the morning to pass stools like “rabbit pellets”
  • bloating

Another possible symptom of diverticular disease is bleeding dark purple blood from your rectum (back passage). This usually occurs after diarrhoea-like cramping pain, and often leads to hospital admission, but fortunately this is an uncommon complication.

Diverticular disease does not cause weight loss, so if you are losing weight, seeing blood in your stools or experiencing frequent bowel changes, see your GP.

Diverticulitis

Diverticulitis shares most of the symptoms of diverticular disease (see above). However, the pain associated with diverticulitis is constant and severe, rather than intermittent. It is most likely to occur if you have previously had symptoms of diverticular disease, and develops over a day or two.

Other symptoms of diverticulitis can include:

  • a high temperature (fever) of 38C (100.4F) or above
  • a general feeling of being tired and unwell
  • feeling sick (nausea) or being sick (vomiting)

The pain usually starts below your belly button, before moving to the lower left-hand side of your abdomen.

In Asian people, the pain may move to the lower right-hand side of your abdomen. This is because East Asian people tend to develop diverticula in a different part of their colon for genetic reasons.

When to seek medical advice

Contact your GP as soon as possible if you think you have symptoms of diverticulitis.

If you have symptoms of diverticular disease and the condition has previously been diagnosed, you do not usually need to contact your GP as the symptoms can be treated at home.

Read more about the treatment of diverticular disease.

If you have not been diagnosed with the condition, contact your GP so they can rule out other conditions with similar symptoms, such as:

  • a stomach ulcer
  • pancreatitis
  • cholecystitis
  • bowel cancer

Irritable bowel syndrome (IBS) can also cause similar symptoms to diverticular disease.

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New Drug Shows Promise Against Psoriasis- Drug rehab center

 — Many patients with the skin disease psoriasis showed significant improvement when taking an experimental drug called ixekizumab, according to a late-stage, phase 3 clinical trial.

“The visible effects of psoriasis can have a major and life-ruining impact on people’s confidence and self-esteem,” study leader Chris Griffiths, a professor of dermatology at the University of Manchester in England, said in a university news release.

“What we saw in this trial was not just the physical aspects of the disease clearing up, but people on the new drug also reporting a marked improvement in their quality of life as they felt more confident and suffered less from itching — far more than in the other two groups,” he said.

The trial was funded by drug maker Eli Lilly and included 2,500 patients with moderate-to-severe psoriasis. Half took ixekizumab, while the other half took an inactive placebo or the widely used psoriasis drugetanercept (brand name Enbrel).

After 12 weeks of treatment, 40 percent of those who took the new drug were free of all psoriasis, and more than 90 percent showed improvement, the research team reported recently in The Lancet. About half of the patients showed improvement as early as week four, Griffiths’ team said

Overall, the patients taking ixekizumab had better results than those taking etanercept or the placebo, the research showed.

As the researchers explained, the new drug works by neutralizing the inflammatory effects of a protein believed to be one of the causes of psoriasis.

“The objective for treating psoriasis has been to reduce the visible symptoms. But new drugs are fast showing us that a realistic goal for all patients should be attaining clear skin and this trial very much sets us on that path,” Griffiths said in the news release.

One expert in the United States said the trial results are welcome news.

“This represents another in a series of recent breakthroughs in the treatment of psoriasis,” said Dr. Andrew Alexis, chair of the department of dermatology at Mount Sinai St. Luke’s and Mount Sinai Roosevelt in New York City. “In addition to improving or clearing the visible features of psoriasis, treatment with ixekizumab also resulted in significant improvement in quality of life,” he said.

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How I Explained Sensory Processing Disorder to My 4-Year-Old- Term life

You know that feeling of irritation you get if the tag on the back of your brand new T-shirt is scratching up against your skin? It bothers you until you take the shirt off and take scissors to it, or it drives you to just rip the it off completely, not even bothered if you end up leaving a hole in your new shirt.

What about that pair of socks you thought would fit and feel just right, and half way through your day, you realize they do not feel or fit just right and you are taking them off in a frenzy so you can feel free and breathe.

And then there’s that feeling of having someone put their hand on your back, or trying to hold your hand, wanting to hug you, or even kiss you — it actually hurts you, and you can’t explain it, you just can’t. So you avoid affection because that is your way of protecting yourself from the pain of human touch.

How do I explain this to my beautiful little girl? She’s 4 years old and I didn’t know how to explain it to her. How do I explain it to her in a way she would understand? How do I explain her younger sister has sensory processing disorder?

Zoey is 3 years old, she’s diagnosed with autism, global delay, ADHD, dyspraxia of speech and sensory processing disorder.

That is hard enough for me to understand, never mind my 4-year-old understanding it.

I put it off, and I would say to her that her sister is “different,” and that “different” isn’t bad.

That explained nothing. I was taking the easy way out. She watched as her younger sister had a year of intensive in-home therapies with multiple therapists, all of them with their own special bag of toys, all coming to play with her little sister. My big girl watched, and every day said, “What about me?” I saw the sadness in her eyes, and I watched as my big girl was feeling left out and confused.

She watched as her little sister got so much more attention, and she started to regress herself because “What about me?” was how she felt, and if she was “different” like her little sister then she would get to have all the extra attention and play with all of the therapists’ special toys.

I had no idea what to say, what to do, or how to help them both. I needed to help them both. I took my big girl aside one night and we had a talk in her room, just her and I. I was not sure how this talk was going to go, but it was time to explain it to her in a “different” way.

I looked at my beautiful 4-year-old and I said, “You know how you have “itchies” on your arms and legs (eczema) and they bother you so much that you scratch them until they feel better, but they don’t ever really feel better until Mommy gives you lotion to help?”

My big girl looked up at me and she said, “Yes, those itchies hurt so bad, and I scratch and scratch and it doesn’t feel better until you help me.”

I said, “Yeah, I help you to make those ‘itchies’ not itch so bad. Your ‘itchies’ are on the outside, and we can see them and we know where they are. Well, sissy’s ‘itchies’ are on the inside, and Mommy can’t see them, so I help her by giving her ‘squeezes’ (deep pressure message on her arms and legs) so she doesn’t itch so bad.”

I know that saying she’s “different” wasn’t working for us, and I finally found a way to explain sensory processing disorder to my beautiful big girl.

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